By P. Tonino
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Additional resources for Gastritis and Gastric Cancer - New Insights in Gastroprot., Diag.,Trtmts
2000). Mucins and mucosal protection in the gastrointestinal tract: new prospects for mucins in the pathology of gastrointestinal disease. , Garrido, M. X. (1995). MUC6 apomucin shows a distinct normal tissue distribution that correlates with Lewis antigen expression in the human stomach. 723-734, ISSN 0016-5085 Dekker, J. J. (1990). Covalent oligomerization of rat gastric mucin occurs in the rough endoplasmic reticulum, is N-glycosylation-dependent, and precedes initial O-glycosylation. ; Kato, S.
Immunostaining for CGRP in rat gastric mucosa. Weak (A; only some glandular cells are positive in the mucosa), medium (B; fine granular cytoplasmic staining) and strong (C; predominantly in the basal zone, larger granular staining is observed with higher intensity) CGRP expression (original magnification: 100x). Approach to Role of Capsaicin - Sensitive Afferent Nerves in the Development and Healing in Patients with Chronic Gastritis A 39 B Fig. 9. Immunostaining for SP in rat gastric mucosa. The weakly stained mucosa (A) is almost negative, however, in positive cases; some fine granular immunostaining was observed (B), corresponding to perivascular neural elements (original magnification: 100x).
Pylori bacteria as etiological factors might represent only one of the factors causing chronic gastritis (in term of histology). 4. The immunhistological distribution of TRVP1, CGRP and SP are independent on the chronic gastritis produced by different physical, chemical, bacteriological or immunological agents. It’s true, that the gastric mucosa did not become histologically intact after eradication treatment. These changes in the histological picture in the gastric mucosa (before and after eradication treatment) suggest that the time period (6 weeks) was short for healing the chronic gastritis by eradication treatment.